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Noonan’s Syndrome – Symptoms, Diagnosis and Medications.

Definition

Noonan’s syndrome is a genetic disorder that prevents normal development in various parts of the body. A person can be affected by Noonan syndrome in a wide variety of ways. These include unusual facial characteristics, short stature, heart defects, other physical problems and possible developmental delays.

Noonan’s syndrome

History

Jacqueline Noonan was practicing as a pediatric cardiologist at the University of Iowa when she noticed that children with a rare type of heart defect, valvular pulmonary stenosis, often had a characteristic physical appearance, with short stature, webbed neck, wide spaced eyes, and low-set ears. Both boys and girls were affected. These characteristics were sometimes seen running in families but were not associated with gross chromosomal abnormalities. She studied 833 patients at the congenital heart disease clinic, looking for other congenital abnormalities, and in 1963 presented a paper: “Associated non-cardiac malformations in children with congenital heart disease”. This described 9 children who in addition to congenital heart disease had characteristic facial features, chest deformities and short stature.

Dr. John Opitz, a former student of Dr. Noonan, first began to call the condition “Noonan syndrome” when he saw children who looked like those whom Dr. Noonan had described. Dr. Noonan produced a paper entitled “Hypertelorism with Turner Phenotype” in 1968, and in 1971 at the Symposium of Cardiovascular defects, the name ‘Noonan syndrome’ became officially recognized.

Incidence/prevalence

The incidence is one in 1,000 to 2,500 live births for severe phenotype, but mild cases may be as common as one in 100 live births. Familial recurrence is consistent with an autosomal dominant mode of inheritance, but de novo mutations are more common, accounting for 60% of cases.There is no known predilection by race or sex

Causes of noonan’s syndrome

Noonan syndrome is caused by a genetic mutation. These mutations can occur in multiple genes. Defects in these genes cause the production of proteins that are continually active. Because these genes play a role in the formation of many tissues throughout the body, this constant activation of proteins disrupts the normal process of cell growth and division.

The mutations that cause Noonan syndrome can be:

Autosomal dominant inheritance pattern

Risk factors of noonan’s syndrome

A parent with Noonan syndrome has a 50 percent chance (one chance in two) of passing the defective gene on to his or her child. The child who inherits the defective gene may have fewer or more symptoms than the affected parent.

Complications of noonan’s syndrome

Complications can arise that may require special attention, including:

Symptoms of noonan’s syndrome

Diagnosis and tests

Treatment and medications

As Noonan syndrome stems from a genetic defect, there is no cure. Treatment aims to manage the associated symptoms and complications, so it varies for each individual.

Potential treatments include:

Prevention of noonan’s syndrome

Couples with a family history of Noonan syndrome may want to consider genetic counseling before having children.

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