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Epidermolysis Bullosa (EB) – Definition, Causes, and Treatments.

Definition

Epidermolysis Bullosa (EB) is a group of rare genetic skin conditions, which is characterized by extremely fragile skin and recurrent blister formation, resulting from minor mechanical friction or trauma. The skin has two layers; the outer layer is called the epidermis and the inner layer called the dermis. Normally, there are ‘anchors’ between the two layers that prevent them from moving independently from one another. In people with EB, the two skin layers lack the anchors that hold them together, and any action that creates friction between the layers (like rubbing or pressure) will create blisters and painful sores. Sufferers of EB have compared the sores to third-degree burns.

In many countries, Butterfly Children is a term often used to describe younger patients because their skin is as fragile as a butterfly’s wings. Sometimes, children with the condition are also described as Cotton Wool Babies. And in South America, Crystal Skin Children is the term used.

Types of EB

There are four major types of EB based on the site of blister formations within the skin layers as follows:

Epidermolysis bullosa simplex (EBS)

In this type, blistering occurs in the upper layer of the skin called the epidermis. It is characterized by a lack of adhesion of the skin directly above the basement membrane (the basal layer). EBS usually is inherited in an autosomal dominant manner, meaning that a defective gene inherited from a parent. The blistering may develop either on hands or feet (localized EBS) or all over the body (generalized EBS).

Junctional epidermolysis bullosa (JEB)

In this type, blistering occurs in a skin layer called the lamina lucida within the basement membrane zone, which is situated at the junction between the epidermis (upper layer of the skin) and the dermis (lower layer). JEB is the most severe type of EB. Like EBS, JEB also is inherited in an autosomal recessive manner.

Dystrophic epidermolysis bullosa (DEB)

In this type, blistering occurs in the lamina densa, which is a component of the basement membrane zone between the lamina lucida and the underlying dermis of the skin. DEB is characterized by the scarring of the healed wounds, resulting in contraction of the joints, fusion of the fingers and toes, contraction of the mouth membranes, and narrowing of the esophagus. It may be inherited in a recessive or dominant manner.

 

Kindler syndrome

In this type of EB, blistering may occur at multiple levels within the basement membrane zone, or in skin layers beneath it. This syndrome is rare within this disease and characterized by blisters on the hands and feet, altered skin coloring, and damage to the inner lining of areas such as the mouth, intestines, or eyes.

Epidermolysis bullosa acquisita (EBA)

Another rare type of EB is called epidermolysis bullosa acquisita. EBA is a non-genetic autoimmune disease, caused by the development of antibodies (proteins that attack foreign substances) against type VII collagen, an essential skin protein. An examination of skin blisters will show antibodies deposited in the basement membrane between the epidermis and dermis. This form of EB is very rare, is not inherited, and usually develops during adulthood.

Pathophysiology

EB is caused by mutations involving at least 18 genes encoding structural proteins within keratin intermediate filaments, focal adhesions, desmosome cell junctions, and hemidesmosome attachment complexes, which form the intraepidermal adhesion and dermoepidermal anchoring complex within the basement membrane zone (BMZ) of the skin and mucosae. The molecular aberrations interfere with the functional and structural integrity of the BMZ (which is a highly specialized interface between epithelial cells and the underlying matrix) that is crucial for cell adhesion, proliferation, and differentiation; tissue repair; and barrier function and leads to cell and tissue dehiscence

Causes of Epidermolysis Bullosa

Risk factors

Clinical manifestations

The main symptoms of all types of EB include:

Complications of Epidermolysis Bullosa

A number of serious complications are associated with EB:

Diagnosis and test

The following procedures may be used to diagnose Epidermolysis Bullosa:

Test

Other tests that may be required depending on the clinical situation include:

Treatment and medications

Epidermolysis bullosa (EB) has no cure and its treatment takes several factors into consideration, including areas affected physiological response to healing, infection, and involvement of other organ systems. Treatment aims to:

Medications

Surgery

Surgery to treat this condition may include:

Rehabilitation therapy

Working with a rehabilitation specialist (physical therapist, occupational therapist) can help ease the limitations on motion caused by scarring and shortening of the skin (contracture). A rehabilitation specialist can also give you guidance on the best ways to keep you or your child safe while going about daily activities. Swimming may also be helpful in preserving or regaining mobility.

Home remedies

Follow these guidelines at home:

Prevention of Epidermolysis Bullosa

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